Preliminary experiments point out conolidine might inhibit specific ion channels, lowering neuronal excitability and limiting ache signals. This system is particularly appropriate in neuropathic soreness, the place irregular signaling triggers persistent soreness. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computational d... https://robertt516fwl8.blogofchange.com/profile